Ninety percent of the brain is immune cells, with neurons making up the remaining 10 percent. Brain care and rehabilitation has always been focused on the neurons, yet the latest research shows the brain’s glial cells, or immune cells, are necessary for memory, cognition, synaptic function, neurotransmitter activity, and other vital brain function.

Healthy glial cell activity is vital not only in managing complex chronic health conditions, but also in lowering the risk of Alzheimer’s, Parkinson’s, and other neurodegenerative diseases.

The Neuroinflammation course will discuss the immune system of the brain and different variables that can induce neurological inflammation. These include traumatic brain injury, diet, metabolic disease, Lyme’s disease, viral infections, and environmental triggers.

The clinician will be taught a step-by-step clinical model on how to identify neuroinflammatory patterns and their sources through patient history, physical examination, laboratory tests, and neuroimaging studies.

We will discuss in detail the role of the blood-brain barrier in neuroinflammation, clinical strategies to address sources of neuroinflammation, and tools to dampen brain inflammation.

This course blends a review of the latest scientific articles with clinical strategies to identify and manage patients with brain inflammation.

The Neuroinflammation course overview:

  • The role of the brain’s immune cells, called glial cells, in maintaining the health of the neurons.
  • How memory, cognition, synaptic function, neurotransmitter activity, and other vital brain functions are dependent on healthy glial cell function.
  • How focusing on the neurons but ignoring glial cells explains why functional neurology protocols often worsen symptoms or fail.
  • Why glial cells should be the primary focus in preventing Alzheimer’s, Parkinson’s, and other neurodegenerative diseases.
  • How glial cells that have become pro-inflammatory actually change in shape, anatomy, and function… and what to do about it.
  • Research breakthroughs in what causes glial cells to become pro-inflammatory.
  • Clinical strategies for disarming activated glial cells.
  • How to identify patterns of neuroinflammation on blood tests.
  • How to figure out what is causing neuroinflammation in each patient; it will be different for each.
  • How healthy glial function supports positive neuron plasticity.
  • How to stimulate the neurons to keep glial cells healthy.
  • How gut bacteria directly communicate with glial cells.
  • We only have so many glial cells — how to prevent accelerated degeneration and loss of glial cells and take care of the ones you and your patients have left.